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One of the more interesting aspects is that while the CS exposure depletes the ELF, plasma, and liver GSH levels, the lung tissue GSH levels remain unchanged

These results indicate that IGF1R deficiency itself confers IGF1 resistance

It would be interesting to investigate if increased -glutamylcysteine could explain the drug resistance observed in tumours associated with -glutamyltransferase overexpression 26

An overview of the stomach-stress-organoprotection hypothesis and beneficial effects of BPC-157." Journal of Physiology (Paris)

Efficacy of inhaled N-acetylcysteine monotherapy in patients with early stage idiopathic pulmonary fibrosis

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